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We also observed downregulation of both Cidea , a fatty acid-induced transcriptional coactivator 28 , which contributes to diet-induced obesity and diabetes 29 , and Acot11 , which encodes a medium/long-chain acyl-CoA thioesterase whose induction is implicated in hepatic glucose production and insulin resistance 30

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To buffer the oxidative stress generated by rapid growth, division, and metabolic abnormalities, cancer cells have a higher demand for glutathione than normal cells, which leads homocysteine to favor the trans-sulfuration pathway over methionine regeneration (30)