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proposed that TGFBR1 overexpression aggravates retinal inflammation via microglial ferroptosis, yet a contradictory finding indicates no significant association between ferroptosis-related genes and disease progression, suggesting that immune cell infiltration occurs independently of ferroptosis (45, 156)
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Together, this coordinated lipolytic cascade mobilizes LD-derived FAs for metabolic adaptation under nutrient stress in cancer cells 22,78,79
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