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Although carnitine-induced FAO was not impaired as much as that in SDT-f-DKD rats, l-carnitine supplementation for 6 weeks had protective effects on ectopic lipid accumulation, tubular injury, and kidney fibrosis without affecting systolic BP (Figure 10, A, I, and J), suggesting that salt-sensitive hypertension partially induces carnitine deficiency possibly via reduction in reabsorption and biosynthesis of carnitine