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Additionally, arsenic trioxide (ATO), more recently introduced in APL therapy, induces post-transcriptional modifications in the second B2 domain of PML, resulting in the restoration of nuclear bodies and degradation of PML::RARA.3 Epidemiologically, APL accounts for approximately 5–10 % of all AML cases, with a slight predilection for adults over children and the elderly

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reported the urinary glucose excretion following administration of the SGLT2 inhibitor dapagliflozin at a dose of 1 mg/kg in C57BL/6 mice