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Further investigation demonstrated that Dicer preferentially binds to unacetylated forms of p53, whereas the interaction between Dicer and p53 is dramatically suppressed by stress-induced p53 acetylation at K120 and K164 (Figure 5.C)

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Supporting this hypothesis, a chronic unpredictable mild stress mouse model demonstrated that melatonin effectively restored aquaporin-4 polarization and rectified the compromised glymphatic system observed in this model [215]