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It is possible that high levels of plasma Cu under the condition of atherosclerosis cause cuproptosis of macrophages and other immune cells in the circulation, leading to unbalanced immunological responses in the circulation

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In addition, knockdown of NNT increased cellular and mitochondrial NADH/total NAD(H) in human SK-Hep1 cells under basal and ADP-stimulated conditions compared with control shRNA-transfected cells, whereas cellular and mitochondrial NADPH/total NADP(H) was much lower in NNT-silenced cells, which correlated with an increase in mitochondrial ROS generation (33)