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Therefore, less adhesion formation as well as reversing existing adhesions, the restoration of normal tissue structure and function, suggest that BPC 157 may act with the temporary role of fibrin in healing without adhesions that must be purposefully degraded by the fibrinolytic system (Collen, 1980
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TREM2 also modulates APOE isoform-specific effects: TREM2 deficiency exacerbates microglial Aβ clearance deficits in APOE ε4 mice (274), supporting TREM2-targeted therapies for APOE ε4-associated AD
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