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But initially, in the early stage of diabetes, ROS are unlikely to travel more than a few microns, lasting for very short periods, before being captured and quenched, and therefore the earliest damaged in diabetic retinopathy occurs because of changes initiated intracellularly (113)

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Consistent with this hypothesis, HOXB1 and GATA exhibit seemingly opposite effects on cardiac differentiation: while HOXB1 delays cardiac differentiation, GATA4-5-6 are known for their established cardiogenic effects, with GATA4 being one of three TFs capable of converting fibroblasts into cardiomyocytes (Ieda et al, 2010)