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DNA lesions are in the form of double-strand breaks, and the first molecule that is going to recruit is ATM serine/threonine kinase, which activates p53 protein (tumour suppressor protein) which in turn regulates another protein PUMA (p53 upregulated modulator of apoptosis) and is a pro-apoptotic protein
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Targeting SLC7A11 in EC Existing studies suggest that SLC7A11 is a potential cancer therapeutic target [235]
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