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Upon NF-κB activation by stress signals, pro-inflammatory cytokines (such as IL-1β and TNF-α), or oxidative stress, NF-κB translocates to the nucleus, where it alters the expression of genes involved in neuroinflammation and neural excitability

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Isoflavones also decrease oxidative stress (both in vivo and in vitro), stimulate NO production, improve systemic arterial compliance, and favourably affect salt and water balance, all of which contribute to a protective role against the development of hypertension (125)